ReP USP - Resultado da busca

Artigo de periodico
Sex- and age-specific respiratory alterations induced by prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 in rats (2023)
Patrone, Luís Gustavo A.; Ferrari, Gustavo Duarte ; Silva, Rodrigo Moreira da ; Alberici, Luciane Carla ; Lopes, Norberto Peporine ; Stabile, Angelita Maria ; Klein, Wilfried ; Bícego, Kênia C.; Gargaglioni, Luciane H.
Fonte: British Journal of Pharmacology. Unidades: FCFRP, EERP, FFCLRP
Assuntos: RECEPTORES, CANABINOIDES, GRAVIDEZ, TRONCO CEREBRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PATRONE, Luís Gustavo A. et al. Sex- and age-specific respiratory alterations induced by prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 in rats. British Journal of Pharmacology, v. 180, n. 13, p. 1-24, 2023Tradução . . Disponível em: https://doi.org/10.1111/bph.16044. Acesso em: 30 abr. 2024.
APA
Patrone, L. G. A., Ferrari, G. D., Silva, R. M. da, Alberici, L. C., Lopes, N. P., Stabile, A. M., et al. (2023). Sex- and age-specific respiratory alterations induced by prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 in rats. British Journal of Pharmacology, 180( 13), 1-24. doi:10.1111/bph.16044
NLM
Patrone LGA, Ferrari GD, Silva RM da, Alberici LC, Lopes NP, Stabile AM, Klein W, Bícego KC, Gargaglioni LH. Sex- and age-specific respiratory alterations induced by prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 in rats [Internet]. British Journal of Pharmacology. 2023 ; 180( 13): 1-24.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/bph.16044
Vancouver
Patrone LGA, Ferrari GD, Silva RM da, Alberici LC, Lopes NP, Stabile AM, Klein W, Bícego KC, Gargaglioni LH. Sex- and age-specific respiratory alterations induced by prenatal exposure to the cannabinoid receptor agonist WIN 55,212-2 in rats [Internet]. British Journal of Pharmacology. 2023 ; 180( 13): 1-24.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/bph.16044
Artigo de periodico
IL-6 deletion decreased REV-ERBα protein and influenced autophagy and mitochondrial markers in the skeletal muscle after acute exercise (2022)
Pinto, Ana Paula ; Muñoz, Vitor Rosetto ; Rocha, Alisson Luiz da ; Rovina, Rafael Lemes ; Ferrari, Gustavo Duarte ; Alberici, Luciane Carla ; Simabuco, Fernando Moreira; Teixeira, Giovana Rampazzo; Pauli, José Rodrigo; Moura, Leandro Pereira de; Cintra, Dennys Esper; Ropelle, Eduardo Rochete; Freitas, Ellen Cristini de ; Rivas, Donato A.; Silva, Adelino Sanchez Ramos da
Fonte: Frontiers in Immunology. Unidades: FCFRP, EEFERP, FMRP
Assuntos: DELEÇÃO DE GENES, MITOCÔNDRIAS, BIOMARCADORES, INTERLEUCINA 6, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINTO, Ana Paula et al. IL-6 deletion decreased REV-ERBα protein and influenced autophagy and mitochondrial markers in the skeletal muscle after acute exercise. Frontiers in Immunology, v. 13, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.953272. Acesso em: 30 abr. 2024.
APA
Pinto, A. P., Muñoz, V. R., Rocha, A. L. da, Rovina, R. L., Ferrari, G. D., Alberici, L. C., et al. (2022). IL-6 deletion decreased REV-ERBα protein and influenced autophagy and mitochondrial markers in the skeletal muscle after acute exercise. Frontiers in Immunology, 13, 1-13. doi:10.3389/fimmu.2022.953272
NLM
Pinto AP, Muñoz VR, Rocha AL da, Rovina RL, Ferrari GD, Alberici LC, Simabuco FM, Teixeira GR, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Freitas EC de, Rivas DA, Silva ASR da. IL-6 deletion decreased REV-ERBα protein and influenced autophagy and mitochondrial markers in the skeletal muscle after acute exercise [Internet]. Frontiers in Immunology. 2022 ; 13 1-13.[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2022.953272
Vancouver
Pinto AP, Muñoz VR, Rocha AL da, Rovina RL, Ferrari GD, Alberici LC, Simabuco FM, Teixeira GR, Pauli JR, Moura LP de, Cintra DE, Ropelle ER, Freitas EC de, Rivas DA, Silva ASR da. IL-6 deletion decreased REV-ERBα protein and influenced autophagy and mitochondrial markers in the skeletal muscle after acute exercise [Internet]. Frontiers in Immunology. 2022 ; 13 1-13.[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fimmu.2022.953272
Artigo de periodico
Neural Infection by Oropouche Virus in Adult Human Brain Slices Induces an Inflammatory and Toxic Response (2021)
Almeida, Glaucia Maria de; Souza, Juliano de Paula; Mendes, Niele Dias; Pontelli, Marjorie Cornejo; Pinheiro, Nathalia Reges; Nogueira, Giovanna Orlovski; Cardoso, Ricardo S.; Paiva, Isadora Marques; Ferrari, Gustavo Duarte ; Veras, Flávio Protásio; Cunha, Fernando de Queiroz ; Horta-Júnior, José de Anchieta de Castro e; Alberici, Luciane Carla; Cunha, Thiago Mattar; Podolsky-Gondim, Guilherme Gozzoli ; Neder, Luciano; Arruda, Eurico; Sebollela, Adriano Silva
Fonte: Frontiers in Neuroscience. Unidades: FCFRP, FMRP, IB
Assuntos: ARBOVÍRUS, ENCEFALITE VIRAL, CÉREBRO, DOENÇAS CEREBRAIS, RNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Glaucia Maria de et al. Neural Infection by Oropouche Virus in Adult Human Brain Slices Induces an Inflammatory and Toxic Response. Frontiers in Neuroscience, v. 15, 2021Tradução . . Disponível em: https://doi.org/10.3389/fnins.2021.674576. Acesso em: 30 abr. 2024.
APA
Almeida, G. M. de, Souza, J. de P., Mendes, N. D., Pontelli, M. C., Pinheiro, N. R., Nogueira, G. O., et al. (2021). Neural Infection by Oropouche Virus in Adult Human Brain Slices Induces an Inflammatory and Toxic Response. Frontiers in Neuroscience, 15. doi:10.3389/fnins.2021.674576
NLM
Almeida GM de, Souza J de P, Mendes ND, Pontelli MC, Pinheiro NR, Nogueira GO, Cardoso RS, Paiva IM, Ferrari GD, Veras FP, Cunha F de Q, Horta-Júnior J de A de C e, Alberici LC, Cunha TM, Podolsky-Gondim GG, Neder L, Arruda E, Sebollela AS. Neural Infection by Oropouche Virus in Adult Human Brain Slices Induces an Inflammatory and Toxic Response [Internet]. Frontiers in Neuroscience. 2021 ; 15[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fnins.2021.674576
Vancouver
Almeida GM de, Souza J de P, Mendes ND, Pontelli MC, Pinheiro NR, Nogueira GO, Cardoso RS, Paiva IM, Ferrari GD, Veras FP, Cunha F de Q, Horta-Júnior J de A de C e, Alberici LC, Cunha TM, Podolsky-Gondim GG, Neder L, Arruda E, Sebollela AS. Neural Infection by Oropouche Virus in Adult Human Brain Slices Induces an Inflammatory and Toxic Response [Internet]. Frontiers in Neuroscience. 2021 ; 15[citado 2024 abr. 30 ] Available from: https://doi.org/10.3389/fnins.2021.674576
Artigo de periodico
The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition (2021)
Queiroz, André L.; Lessard, Sarah J.; Ouchida, Amanda T.; Araujo, Hygor N.; Gonçalves, Dawit A.; Guimarães, Dimitrius Santiago Passos Simões Fróes; Teodoro, Bruno G.; So, Kawai; Espreáfico, Enilza Maria ; Hirshman, Michael F.; Alberici, Luciane Carla ; Kettelhut, Isis do Carmo ; Goodyear, Laurie J.; Silveira, Leonardo R.
Fonte: Molecular Metabolism. Unidades: FMRP, FCFRP
Assuntos: MICRORNAS, MÚSCULO ESQUELÉTICO, MITOCÔNDRIAS, REGULAÇÃO GÊNICA, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUEIROZ, André L. et al. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, v. 51, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2021.101226. Acesso em: 30 abr. 2024.
APA
Queiroz, A. L., Lessard, S. J., Ouchida, A. T., Araujo, H. N., Gonçalves, D. A., Guimarães, D. S. P. S. F., et al. (2021). The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, 51. doi:10.1016/j.molmet.2021.101226
NLM
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Vancouver
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Artigo de periodico
Taurine supplementation associated with exercise increases mitochondrial activity and fatty acid oxidation gene expression in the subcutaneous white adipose tissue of obese women (2021)
Carvalho, Flávia Giolo de ; Brandão, Camila Fernanda Costa e Cunha Moraes; Batitucci, Gabriela; Souza, Anderson de Oliveira; Ferrari, Gustavo Duarte ; Alberici, Luciane Carla; Muñoz, Vitor Rosetto; Pauli, José Rodrigo; Moura, Leandro Pereira de; Ropelle, Eduardo Rochete; Silva, Adelino Sanchez Ramos da ; Junqueira-Franco, Márcia Varella Morandi; Marchini, Julio Sergio ; Freitas, Ellen Cristini de
Fonte: Clinical Nutrition. Unidades: FCFRP, EEFERP, FMRP
Assuntos: EXERCÍCIO FÍSICO, METABOLISMO, MITOCÔNDRIAS, TECIDO ADIPOSO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Flávia Giolo de et al. Taurine supplementation associated with exercise increases mitochondrial activity and fatty acid oxidation gene expression in the subcutaneous white adipose tissue of obese women. Clinical Nutrition, v. 40, p. 2180-2187, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.clnu.2020.09.044. Acesso em: 30 abr. 2024.
APA
Carvalho, F. G. de, Brandão, C. F. C. e C. M., Batitucci, G., Souza, A. de O., Ferrari, G. D., Alberici, L. C., et al. (2021). Taurine supplementation associated with exercise increases mitochondrial activity and fatty acid oxidation gene expression in the subcutaneous white adipose tissue of obese women. Clinical Nutrition, 40, 2180-2187. doi:10.1016/j.clnu.2020.09.044
NLM
Carvalho FG de, Brandão CFC e CM, Batitucci G, Souza A de O, Ferrari GD, Alberici LC, Muñoz VR, Pauli JR, Moura LP de, Ropelle ER, Silva ASR da, Junqueira-Franco MVM, Marchini JS, Freitas EC de. Taurine supplementation associated with exercise increases mitochondrial activity and fatty acid oxidation gene expression in the subcutaneous white adipose tissue of obese women [Internet]. Clinical Nutrition. 2021 ; 40 2180-2187.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.clnu.2020.09.044
Vancouver
Carvalho FG de, Brandão CFC e CM, Batitucci G, Souza A de O, Ferrari GD, Alberici LC, Muñoz VR, Pauli JR, Moura LP de, Ropelle ER, Silva ASR da, Junqueira-Franco MVM, Marchini JS, Freitas EC de. Taurine supplementation associated with exercise increases mitochondrial activity and fatty acid oxidation gene expression in the subcutaneous white adipose tissue of obese women [Internet]. Clinical Nutrition. 2021 ; 40 2180-2187.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.clnu.2020.09.044
Artigo de periodico
Acetyl‐CoA‐driven respiration in frozen muscle contributes to the diagnosis of mitochondrial disease (2021)
Reis, Felippe Henrique Zuccolotto dos; Andrião-Escarso, Sílvia Helena ; Araujo, Jackeline Souza; Espreáfico, Enilza Maria ; Alberici, Luciane Carla ; Sobreira, Claudia Ferreira da Rosa
Fonte: European Journal of Clinical Investigation. Unidades: FMRP, FCFRP
Assuntos: BIÓPSIA, RESPIRAÇÃO CELULAR, TÉCNICAS E PROCEDIMENTOS DE LABORATÓRIO, CRIOPRESERVAÇÃO, TRANSPORTE DE ELÉTRONS, MÚSCULO ESQUELÉTICO, CONSUMO DE OXIGÊNIO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Felippe Henrique Zuccolotto dos et al. Acetyl‐CoA‐driven respiration in frozen muscle contributes to the diagnosis of mitochondrial disease. European Journal of Clinical Investigation, v. 51, n. 9, 2021Tradução . . Disponível em: https://doi.org/10.1111/eci.13574. Acesso em: 30 abr. 2024.
APA
Reis, F. H. Z. dos, Andrião-Escarso, S. H., Araujo, J. S., Espreáfico, E. M., Alberici, L. C., & Sobreira, C. F. da R. (2021). Acetyl‐CoA‐driven respiration in frozen muscle contributes to the diagnosis of mitochondrial disease. European Journal of Clinical Investigation, 51( 9). doi:10.1111/eci.13574
NLM
Reis FHZ dos, Andrião-Escarso SH, Araujo JS, Espreáfico EM, Alberici LC, Sobreira CF da R. Acetyl‐CoA‐driven respiration in frozen muscle contributes to the diagnosis of mitochondrial disease [Internet]. European Journal of Clinical Investigation. 2021 ; 51( 9):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/eci.13574
Vancouver
Reis FHZ dos, Andrião-Escarso SH, Araujo JS, Espreáfico EM, Alberici LC, Sobreira CF da R. Acetyl‐CoA‐driven respiration in frozen muscle contributes to the diagnosis of mitochondrial disease [Internet]. European Journal of Clinical Investigation. 2021 ; 51( 9):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1111/eci.13574
Artigo de periodico
Endogenous galectin-3 is required for skeletal muscle repair (2021)
Cerri, Daniel Giuliano; Rodrigues, Lilian Cataldi ; Alves, Vani Maria; Machado, Juliano; Bastos, Victor Alexandre Félix; Kettelhut, Isis do Carmo ; Alberici, Luciane Carla; Costa, Maria Cristina R; Stowell, Sean R; Cummings, Richard D; Dias-Baruffi, Marcelo
Fonte: Glycobiology. Unidades: FCFRP, FMRP
Assuntos: MIOBLASTOS, BÁRIO, CLORETO, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERRI, Daniel Giuliano et al. Endogenous galectin-3 is required for skeletal muscle repair. Glycobiology, v. 31, n. 10, p. 1295-1307, 2021Tradução . . Disponível em: https://doi.org/10.1093/glycob/cwab071. Acesso em: 30 abr. 2024.
APA
Cerri, D. G., Rodrigues, L. C., Alves, V. M., Machado, J., Bastos, V. A. F., Kettelhut, I. do C., et al. (2021). Endogenous galectin-3 is required for skeletal muscle repair. Glycobiology, 31( 10), 1295-1307. doi:10.1093/glycob/cwab071
NLM
Cerri DG, Rodrigues LC, Alves VM, Machado J, Bastos VAF, Kettelhut I do C, Alberici LC, Costa MCR, Stowell SR, Cummings RD, Dias-Baruffi M. Endogenous galectin-3 is required for skeletal muscle repair [Internet]. Glycobiology. 2021 ; 31( 10): 1295-1307.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1093/glycob/cwab071
Vancouver
Cerri DG, Rodrigues LC, Alves VM, Machado J, Bastos VAF, Kettelhut I do C, Alberici LC, Costa MCR, Stowell SR, Cummings RD, Dias-Baruffi M. Endogenous galectin-3 is required for skeletal muscle repair [Internet]. Glycobiology. 2021 ; 31( 10): 1295-1307.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1093/glycob/cwab071
Artigo de periodico
A freeze-and-thaw-induced fragment of the microtubule-associated protein tau in rat brain extracts: implications for the biochemical assessment of neurotoxicity (2021)
Vasconcelos, Israel Costa; Campos, Raquel Maria de; Schwaemmle, Hanna; Masson, Ana Paula; Ferrari, Gustavo Duarte ; Alberici, Luciane Carla; Faça, Vitor Marcel ; Garcia-Cairasco, Norberto ; Sebollela, Adriano Silva
Fonte: Bioscience Reports. Unidades: FMRP, FCFRP
Assuntos: DOENÇA DE ALZHEIMER, CÉREBRO, CRIOPRESERVAÇÃO, IMUNOHISTOQUÍMICA, METABOLISMO DE PROTEÍNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VASCONCELOS, Israel Costa et al. A freeze-and-thaw-induced fragment of the microtubule-associated protein tau in rat brain extracts: implications for the biochemical assessment of neurotoxicity. Bioscience Reports, v. 41, n. 3, p. 1-11, 2021Tradução . . Disponível em: https://doi.org/10.1042/BSR20203980. Acesso em: 30 abr. 2024.
APA
Vasconcelos, I. C., Campos, R. M. de, Schwaemmle, H., Masson, A. P., Ferrari, G. D., Alberici, L. C., et al. (2021). A freeze-and-thaw-induced fragment of the microtubule-associated protein tau in rat brain extracts: implications for the biochemical assessment of neurotoxicity. Bioscience Reports, 41( 3), 1-11. doi:10.1042/BSR20203980
NLM
Vasconcelos IC, Campos RM de, Schwaemmle H, Masson AP, Ferrari GD, Alberici LC, Faça VM, Garcia-Cairasco N, Sebollela AS. A freeze-and-thaw-induced fragment of the microtubule-associated protein tau in rat brain extracts: implications for the biochemical assessment of neurotoxicity [Internet]. Bioscience Reports. 2021 ; 41( 3): 1-11.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/BSR20203980
Vancouver
Vasconcelos IC, Campos RM de, Schwaemmle H, Masson AP, Ferrari GD, Alberici LC, Faça VM, Garcia-Cairasco N, Sebollela AS. A freeze-and-thaw-induced fragment of the microtubule-associated protein tau in rat brain extracts: implications for the biochemical assessment of neurotoxicity [Internet]. Bioscience Reports. 2021 ; 41( 3): 1-11.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1042/BSR20203980
Artigo de periodico
Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S (2020)
Azevedo, Lara Ferreira ; Carneiro, Maria Fernanda Hornos ; Dechandt, Carlos Roberto Porto ; Cassoli, Juliana Silva ; Alberici, Luciane Carla ; Barbosa Junior, Fernando
Fonte: Environmental Research. Unidade: FCFRP
Assuntos: HOMEOSTASE, FÍGADO, PROTEÔMICA, FENÓIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AZEVEDO, Lara Ferreira et al. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S. Environmental Research, v. 182, p. 109080, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.envres.2019.109080. Acesso em: 30 abr. 2024.
APA
Azevedo, L. F., Carneiro, M. F. H., Dechandt, C. R. P., Cassoli, J. S., Alberici, L. C., & Barbosa Junior, F. (2020). Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S. Environmental Research, 182, 109080. doi:10.1016/j.envres.2019.109080
NLM
Azevedo LF, Carneiro MFH, Dechandt CRP, Cassoli JS, Alberici LC, Barbosa Junior F. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S [Internet]. Environmental Research. 2020 ; 182 109080.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.envres.2019.109080
Vancouver
Azevedo LF, Carneiro MFH, Dechandt CRP, Cassoli JS, Alberici LC, Barbosa Junior F. Global liver proteomic analysis of Wistar rats chronically exposed to low-levels of bisphenol A and S [Internet]. Environmental Research. 2020 ; 182 109080.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.envres.2019.109080
Artigo de periodico
Differential expression of antioxidant system genes in honey bee (Apis mellifera L.) caste development mitigates ROS-mediated oxidative damage in queen larvae (2020)
Santos, Douglas Elias ; Souza, Anderson de Oliveira ; Tibério, Gustavo Jacomini; Alberici, Luciane Carla ; Hartfelder, Klaus
Fonte: Genetics and Molecular Biology. Unidades: FCFRP, FMRP
Assuntos: INSETOS SOCIAIS, ESPÉCIES REATIVAS DE OXIGÊNIO, ABELHAS, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Douglas Elias et al. Differential expression of antioxidant system genes in honey bee (Apis mellifera L.) caste development mitigates ROS-mediated oxidative damage in queen larvae. Genetics and Molecular Biology, v. 43, n. 4, 2020Tradução . . Disponível em: https://doi.org/10.1590/1678-4685-GMB-2020-0173. Acesso em: 30 abr. 2024.
APA
Santos, D. E., Souza, A. de O., Tibério, G. J., Alberici, L. C., & Hartfelder, K. (2020). Differential expression of antioxidant system genes in honey bee (Apis mellifera L.) caste development mitigates ROS-mediated oxidative damage in queen larvae. Genetics and Molecular Biology, 43( 4). doi:10.1590/1678-4685-GMB-2020-0173
NLM
Santos DE, Souza A de O, Tibério GJ, Alberici LC, Hartfelder K. Differential expression of antioxidant system genes in honey bee (Apis mellifera L.) caste development mitigates ROS-mediated oxidative damage in queen larvae [Internet]. Genetics and Molecular Biology. 2020 ; 43( 4):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/1678-4685-GMB-2020-0173
Vancouver
Santos DE, Souza A de O, Tibério GJ, Alberici LC, Hartfelder K. Differential expression of antioxidant system genes in honey bee (Apis mellifera L.) caste development mitigates ROS-mediated oxidative damage in queen larvae [Internet]. Genetics and Molecular Biology. 2020 ; 43( 4):[citado 2024 abr. 30 ] Available from: https://doi.org/10.1590/1678-4685-GMB-2020-0173
Artigo de periodico
RANKL induces beige adipocyte differentiation in preadipocytes (2020)
Matsuo, Flávia Sayuri; Araújo, Paulo Henrique Cavalcanti de; Mota, Ryerson Fonseca; Carvalho, Ana Júlia Rossoni; Queiroz, Mariana Santos de; Almeida, Beatriz Baldo de; Ferreira, Karen Cristine de Oliveira Santos ; Metzner, Rodrigo Jair Morandi; Ferrari, Gustavo Duarte ; Alberici, Luciane Carla; Osako, Mariana Kiomy
Fonte: American Journal of Physiology-Endocrinology and Metabolism. Unidades: FCFRP, FMRP
Assuntos: GANHO DE PESO, CONSUMO DE OXIGÊNIO, OBESIDADE, TECIDO ADIPOSO, METABOLISMO ENERGÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MATSUO, Flávia Sayuri et al. RANKL induces beige adipocyte differentiation in preadipocytes. American Journal of Physiology-Endocrinology and Metabolism, v. 318, n. 6, p. E866-E877, 2020Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00397.2019. Acesso em: 30 abr. 2024.
APA
Matsuo, F. S., Araújo, P. H. C. de, Mota, R. F., Carvalho, A. J. R., Queiroz, M. S. de, Almeida, B. B. de, et al. (2020). RANKL induces beige adipocyte differentiation in preadipocytes. American Journal of Physiology-Endocrinology and Metabolism, 318( 6), E866-E877. doi:10.1152/ajpendo.00397.2019
NLM
Matsuo FS, Araújo PHC de, Mota RF, Carvalho AJR, Queiroz MS de, Almeida BB de, Ferreira KC de OS, Metzner RJM, Ferrari GD, Alberici LC, Osako MK. RANKL induces beige adipocyte differentiation in preadipocytes [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2020 ; 318( 6): E866-E877.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpendo.00397.2019
Vancouver
Matsuo FS, Araújo PHC de, Mota RF, Carvalho AJR, Queiroz MS de, Almeida BB de, Ferreira KC de OS, Metzner RJM, Ferrari GD, Alberici LC, Osako MK. RANKL induces beige adipocyte differentiation in preadipocytes [Internet]. American Journal of Physiology-Endocrinology and Metabolism. 2020 ; 318( 6): E866-E877.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1152/ajpendo.00397.2019
Artigo de periodico
Impairment of PGC-1α-mediated mitochondrial biogenesis precedes mitochondrial dysfunction and Alzheimer's pathology in the 3xTg mouse model of Alzheimer's disease (2020)
Singulani, Monique Patricio ; Pereira, Carolina Parga Martins; Ferreira, Ana Flávia Fernandes ; Garcia, Priscila Crespo ; Ferrari, Gustavo Duarte; Alberici, Luciane Carla ; Britto, Luiz Roberto Giorgetti de
Fonte: Experimental Gerontology. Unidades: FCFRP, ICB, FMRP
Assuntos: FISIOLOGIA, MITOCÔNDRIAS, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS, CAMUNDONGOS, TRANSCRIÇÃO GÊNICA, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SINGULANI, Monique Patricio et al. Impairment of PGC-1α-mediated mitochondrial biogenesis precedes mitochondrial dysfunction and Alzheimer's pathology in the 3xTg mouse model of Alzheimer's disease. Experimental Gerontology, v. 133, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2020.110882. Acesso em: 30 abr. 2024.
APA
Singulani, M. P., Pereira, C. P. M., Ferreira, A. F. F., Garcia, P. C., Ferrari, G. D., Alberici, L. C., & Britto, L. R. G. de. (2020). Impairment of PGC-1α-mediated mitochondrial biogenesis precedes mitochondrial dysfunction and Alzheimer's pathology in the 3xTg mouse model of Alzheimer's disease. Experimental Gerontology, 133, 10 . doi:10.1016/j.exger.2020.110882
NLM
Singulani MP, Pereira CPM, Ferreira AFF, Garcia PC, Ferrari GD, Alberici LC, Britto LRG de. Impairment of PGC-1α-mediated mitochondrial biogenesis precedes mitochondrial dysfunction and Alzheimer's pathology in the 3xTg mouse model of Alzheimer's disease [Internet]. Experimental Gerontology. 2020 ; 133 10 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.exger.2020.110882
Vancouver
Singulani MP, Pereira CPM, Ferreira AFF, Garcia PC, Ferrari GD, Alberici LC, Britto LRG de. Impairment of PGC-1α-mediated mitochondrial biogenesis precedes mitochondrial dysfunction and Alzheimer's pathology in the 3xTg mouse model of Alzheimer's disease [Internet]. Experimental Gerontology. 2020 ; 133 10 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.exger.2020.110882
Artigo de periodico
One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state (2020)
Santos, Jonathas Rodrigo dos; Bortolanza, Mariza; Ferrari, Gustavo Duarte ; Lanfredi, Guilherme Pauperio ; Nascimento, Glauce Crivelaro do ; Azzolini, Ana Elisa Caleiro Seixas ; Del Bel, Elaine Aparecida ; Campos, Alline Cristina de; Faça, Vitor Marcel ; Vulczak, Anderson; Alberici, Luciane Carla
Fonte: Antioxidants. Unidades: FMRP, FORP, FCFRP
Assuntos: EXERCÍCIO FÍSICO, ESTRESSE OXIDATIVO, NEUROGENÉTICA, CÉREBRO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTOS, Jonathas Rodrigo dos et al. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state. Antioxidants, v. 9, n. 5, 2020Tradução . . Disponível em: https://doi.org/10.3390/antiox9050445. Acesso em: 30 abr. 2024.
APA
Santos, J. R. dos, Bortolanza, M., Ferrari, G. D., Lanfredi, G. P., Nascimento, G. C. do, Azzolini, A. E. C. S., et al. (2020). One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state. Antioxidants, 9( 5). doi:10.3390/antiox9050445
NLM
Santos JR dos, Bortolanza M, Ferrari GD, Lanfredi GP, Nascimento GC do, Azzolini AECS, Del Bel EA, Campos AC de, Faça VM, Vulczak A, Alberici LC. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state [Internet]. Antioxidants. 2020 ; 9( 5):[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/antiox9050445
Vancouver
Santos JR dos, Bortolanza M, Ferrari GD, Lanfredi GP, Nascimento GC do, Azzolini AECS, Del Bel EA, Campos AC de, Faça VM, Vulczak A, Alberici LC. One-week high-intensity interval training increases hippocampal plasticity and mitochondrial content without changes in redox state [Internet]. Antioxidants. 2020 ; 9( 5):[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/antiox9050445
Artigo de periodico
Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss (2020)
Amorim, Mateus Ramos; Moreira, Diego Azevedo; Santos, Bruna Maitan; Ferrari, Gustavo Duarte; Nogueira, Jonatas Evandro ; Deus, Júnia Lara de; Alberici, Luciane Carla; Branco, Luiz Guilherme de Siqueira
Fonte: The Journal of Physiology. Unidades: EEFERP, FCFRP, FORP, FMRP
Assuntos: HIPOTERMIA, LIPOPOLISSACARÍDEOS, PRESSÃO SANGUÍNEA, TEMPERATURA CORPORAL, HIPERTENSÃO, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMORIM, Mateus Ramos et al. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss. The Journal of Physiology, v. 598, n. 20, p. 4663-4680, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP280321. Acesso em: 30 abr. 2024.
APA
Amorim, M. R., Moreira, D. A., Santos, B. M., Ferrari, G. D., Nogueira, J. E., Deus, J. L. de, et al. (2020). Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss. The Journal of Physiology, 598( 20), 4663-4680. doi:10.1113/JP280321
NLM
Amorim MR, Moreira DA, Santos BM, Ferrari GD, Nogueira JE, Deus JL de, Alberici LC, Branco LG de S. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss [Internet]. The Journal of Physiology. 2020 ; 598( 20): 4663-4680.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1113/JP280321
Vancouver
Amorim MR, Moreira DA, Santos BM, Ferrari GD, Nogueira JE, Deus JL de, Alberici LC, Branco LG de S. Increased lipopolysaccharide‐induced hypothermia in neurogenic hypertension is caused by reduced hypothalamic PGE₂ production and increased heat loss [Internet]. The Journal of Physiology. 2020 ; 598( 20): 4663-4680.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1113/JP280321
Artigo de periodico
Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles (2020)
Adeyemi, Joseph A.; Machado, Ana Rita Thomazela ; Ogunjimi, Abayomi T.; Alberici, Luciane Carla; Antunes, Lusânia Maria Greggi ; Barbosa Júnior, Fernando
Fonte: Ecotoxicology and Environmental Safety. Unidade: FCFRP
Assuntos: CARBONO, QUÍMICA, COBRE, POLUIÇÃO AMBIENTAL, MITOCÔNDRIAS, NANOPARTÍCULAS, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ADEYEMI, Joseph A. et al. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles. Ecotoxicology and Environmental Safety, v. 189, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.ecoenv.2019.109982. Acesso em: 30 abr. 2024.
APA
Adeyemi, J. A., Machado, A. R. T., Ogunjimi, A. T., Alberici, L. C., Antunes, L. M. G., & Barbosa Júnior, F. (2020). Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles. Ecotoxicology and Environmental Safety, 189. doi:10.1016/j.ecoenv.2019.109982
NLM
Adeyemi JA, Machado ART, Ogunjimi AT, Alberici LC, Antunes LMG, Barbosa Júnior F. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles [Internet]. Ecotoxicology and Environmental Safety. 2020 ; 189[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.ecoenv.2019.109982
Vancouver
Adeyemi JA, Machado ART, Ogunjimi AT, Alberici LC, Antunes LMG, Barbosa Júnior F. Cytotoxicity, mutagenicity, oxidative stress and mitochondrial impairment in human hepatoma (HepG2) cells exposed to copper oxide, copper-iron oxide and carbon nanoparticles [Internet]. Ecotoxicology and Environmental Safety. 2020 ; 189[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.ecoenv.2019.109982
Artigo de periodico
UCP2 expression is negatively correlated with and body fat mass after combined physical training: a pilot study (2020)
Brandão, Camila Fernanda Cunha ; Carvalho, Flávia Giolo de ; Nicoletti, Carolina Ferreira ; Junqueira-Franco, Márcia Varella Morandi; Couto-Lima, Carlos Antônio; Souza, Anderson de Oliveira; Santibáñez, Rocío Paulina San Martín; Freitas, Ellen Cristini de ; Alberici, Luciane Carla ; Nonino, Carla Barbosa ; Marchini, Julio Sergio
Fonte: Nutrire. Unidades: FMRP, EEFERP, FCFRP
Assuntos: TREINAMENTO FÍSICO, TERMOGÊNESE, CALORIAS, EXPRESSÃO GÊNICA, OBESIDADE, MULHERES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRANDÃO, Camila Fernanda Cunha et al. UCP2 expression is negatively correlated with and body fat mass after combined physical training: a pilot study. Nutrire, v. 45, n. 1, p. 1-7, 2020Tradução . . Disponível em: https://doi.org/10.1186/s41110-020-00116-2. Acesso em: 30 abr. 2024.
APA
Brandão, C. F. C., Carvalho, F. G. de, Nicoletti, C. F., Junqueira-Franco, M. V. M., Couto-Lima, C. A., Souza, A. de O., et al. (2020). UCP2 expression is negatively correlated with and body fat mass after combined physical training: a pilot study. Nutrire, 45( 1), 1-7. doi:10.1186/s41110-020-00116-2
NLM
Brandão CFC, Carvalho FG de, Nicoletti CF, Junqueira-Franco MVM, Couto-Lima CA, Souza A de O, Santibáñez RPSM, Freitas EC de, Alberici LC, Nonino CB, Marchini JS. UCP2 expression is negatively correlated with and body fat mass after combined physical training: a pilot study [Internet]. Nutrire. 2020 ; 45( 1): 1-7.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1186/s41110-020-00116-2
Vancouver
Brandão CFC, Carvalho FG de, Nicoletti CF, Junqueira-Franco MVM, Couto-Lima CA, Souza A de O, Santibáñez RPSM, Freitas EC de, Alberici LC, Nonino CB, Marchini JS. UCP2 expression is negatively correlated with and body fat mass after combined physical training: a pilot study [Internet]. Nutrire. 2020 ; 45( 1): 1-7.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1186/s41110-020-00116-2
Artigo de periodico
Physical exercise protects against mitochondria alterations in the 6-hidroxydopamine rat model of Parkinson’s disease (2020)
Ferreira, Ana Flávia Fernandes ; Binda, Karina Henrique; Singulani, Monique Patricio ; Pereira, Carolina Parga Martins; Ferrari, Gustavo Duarte ; Alberici, Luciane Carla ; Gregório, Caroline Cristiano Real ; Britto, Luiz Roberto Giorgetti de
Fonte: Behavioural Brain Research. Unidades: FCFRP, ICB, FMRP, FM
Assuntos: FISIOLOGIA, DOENÇA DE PARKINSON, EXERCÍCIO FÍSICO, RATOS WISTAR, RECEPTORES DOPAMINÉRGICOS, SISTEMA RESPIRATÓRIO, MITOCÔNDRIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Ana Flávia Fernandes et al. Physical exercise protects against mitochondria alterations in the 6-hidroxydopamine rat model of Parkinson’s disease. Behavioural Brain Research, v. 387, p. 11 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2020.112607. Acesso em: 30 abr. 2024.
APA
Ferreira, A. F. F., Binda, K. H., Singulani, M. P., Pereira, C. P. M., Ferrari, G. D., Alberici, L. C., et al. (2020). Physical exercise protects against mitochondria alterations in the 6-hidroxydopamine rat model of Parkinson’s disease. Behavioural Brain Research, 387, 11 . doi:10.1016/j.bbr.2020.112607
NLM
Ferreira AFF, Binda KH, Singulani MP, Pereira CPM, Ferrari GD, Alberici LC, Gregório CCR, Britto LRG de. Physical exercise protects against mitochondria alterations in the 6-hidroxydopamine rat model of Parkinson’s disease [Internet]. Behavioural Brain Research. 2020 ; 387 11 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.bbr.2020.112607
Vancouver
Ferreira AFF, Binda KH, Singulani MP, Pereira CPM, Ferrari GD, Alberici LC, Gregório CCR, Britto LRG de. Physical exercise protects against mitochondria alterations in the 6-hidroxydopamine rat model of Parkinson’s disease [Internet]. Behavioural Brain Research. 2020 ; 387 11 .[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.bbr.2020.112607
Artigo de periodico
Moderate exercise modulates tumor metabolism of triple-negative breast cancer (2020)
Vulczak, Anderson ; Souza, Anderson de Oliveira ; Ferrari, Gustavo Duarte ; Azzolini, Ana Elisa Caleiro Seixas ; Bisson, Gabriela Silva; Alberici, Luciane Carla
Fonte: Cells. Unidades: FCFRP, EERP, FMRP
Assuntos: MITOCÔNDRIAS, NEOPLASIAS MAMÁRIAS, ATIVIDADE FÍSICA, EXERCÍCIO FÍSICO, METABOLISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VULCZAK, Anderson et al. Moderate exercise modulates tumor metabolism of triple-negative breast cancer. Cells, v. 9, n. 3, 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9030628. Acesso em: 30 abr. 2024.
APA
Vulczak, A., Souza, A. de O., Ferrari, G. D., Azzolini, A. E. C. S., Bisson, G. S., & Alberici, L. C. (2020). Moderate exercise modulates tumor metabolism of triple-negative breast cancer. Cells, 9( 3). doi:10.3390/cells9030628
NLM
Vulczak A, Souza A de O, Ferrari GD, Azzolini AECS, Bisson GS, Alberici LC. Moderate exercise modulates tumor metabolism of triple-negative breast cancer [Internet]. Cells. 2020 ; 9( 3):[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells9030628
Vancouver
Vulczak A, Souza A de O, Ferrari GD, Azzolini AECS, Bisson GS, Alberici LC. Moderate exercise modulates tumor metabolism of triple-negative breast cancer [Internet]. Cells. 2020 ; 9( 3):[citado 2024 abr. 30 ] Available from: https://doi.org/10.3390/cells9030628
Artigo de periodico
Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death (2020)
Andreu, Gilberto L. Pardo; Reis, Felipe Henrique Zuccolotto dos ; González-Durruthy, Michael; Hernández, René Delgado; D'Vries, Richard F.; Vanden Berghe, Wim; Alberici, Luciane Carla
Fonte: Toxicology in Vitro. Unidades: FCFRP, FMRP
Assuntos: ANTINEOPLÁSICOS, TOXICOLOGIA, NEOPLASIAS, MITOCÔNDRIAS, CARCINOMA HEPATOCELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ANDREU, Gilberto L. Pardo et al. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death. Toxicology in Vitro, v. 63, p. 1-10, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.tiv.2019.104737. Acesso em: 30 abr. 2024.
APA
Andreu, G. L. P., Reis, F. H. Z. dos, González-Durruthy, M., Hernández, R. D., D'Vries, R. F., Vanden Berghe, W., & Alberici, L. C. (2020). Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death. Toxicology in Vitro, 63, 1-10. doi:10.1016/j.tiv.2019.104737
NLM
Andreu GLP, Reis FHZ dos, González-Durruthy M, Hernández RD, D'Vries RF, Vanden Berghe W, Alberici LC. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death [Internet]. Toxicology in Vitro. 2020 ; 63 1-10.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.tiv.2019.104737
Vancouver
Andreu GLP, Reis FHZ dos, González-Durruthy M, Hernández RD, D'Vries RF, Vanden Berghe W, Alberici LC. Rapanone, a naturally occurring benzoquinone, inhibits mitochondrial respiration and induces HepG2 cell death [Internet]. Toxicology in Vitro. 2020 ; 63 1-10.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1016/j.tiv.2019.104737
Artigo de periodico
Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration (2020)
Catalão, Carlos Henrique Rocha ; Santos Junior, Nilton Nascimento dos ; Costa, Luís Henrique Angenendt da ; Souza, Anderson Oliveira; Cárnio, Evelin Capellari ; Sebollela, Adriano Silva ; Alberici, Luciane Carla ; Rocha, Maria José Alves da
Fonte: Neurotoxicity Research. Unidades: FMRP, EERP, FCFRP, FORP
Assuntos: ENCEFALOPATIA ANIMAL, HIPOCAMPO, CÓRTEX PRÉ-FRONTAL, ASTRÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CATALÃO, Carlos Henrique Rocha et al. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration. Neurotoxicity Research, v. 38, n. 4, p. 871-886, 2020Tradução . . Disponível em: https://doi.org/10.1007/s12640-020-00222-z. Acesso em: 30 abr. 2024.
APA
Catalão, C. H. R., Santos Junior, N. N. dos, Costa, L. H. A. da, Souza, A. O., Cárnio, E. C., Sebollela, A. S., et al. (2020). Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration. Neurotoxicity Research, 38( 4), 871-886. doi:10.1007/s12640-020-00222-z
NLM
Catalão CHR, Santos Junior NN dos, Costa LHA da, Souza AO, Cárnio EC, Sebollela AS, Alberici LC, Rocha MJA da. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 871-886.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1007/s12640-020-00222-z
Vancouver
Catalão CHR, Santos Junior NN dos, Costa LHA da, Souza AO, Cárnio EC, Sebollela AS, Alberici LC, Rocha MJA da. Simvastatin prevents long-term cognitive deficits in sepsis survivor rats by reducing neuroinflammation and neurodegeneration [Internet]. Neurotoxicity Research. 2020 ; 38( 4): 871-886.[citado 2024 abr. 30 ] Available from: https://doi.org/10.1007/s12640-020-00222-z

Unidades USP

ReP

Filtros

Unidades USP

Departamento

Autores

Autores USP

Ano de publicação

Assuntos

Título da fonte

Editora

Nome do evento

País de publicação

Agência de fomento

Indexado em

Afiliação dos autores externos não normalizada

País das instituições de afiliação dos autores externos